| Accession |
PRJCA000243 |
| Title |
MSCs ChIP-seq |
| Relevance |
Model organism |
| Data types |
Epigenomics
|
| Organisms |
Mus musculus
|
| Description |
De novo ASXL1 mutations are found in patients with Bohring-Opitz syndrome (BOS), a disease with severe developmental defects and early childhood fatality. The underlying pathologic mechanisms remain largely unknown. |
| Sample scope |
Multiisolate |
| Release date |
2016-09-10 |
| Grants |
| Agency |
program |
Grant ID |
Grant title |
| National Institutes of Health
|
|
CA172408
|
CA172408
|
| National Institutes of Health
|
|
CA185751
|
CA185751
|
| National Institutes of Health
|
|
HL112294
|
HL112294
|
| National Natural Science Foundation of China (NSFC)
|
|
81270612
|
81270612
|
| Chinese Academy of Sciences (CAS)
|
|
153F11KYSB20150013
|
153F11KYSB20150013
|
|
|
| Submitter |
Cai
ying
Zhu (Zhucy5@163.com)
|
| Organization |
State Key Laboratory of Experimental Hematology ,Chinese |
| Submission date |
2016-06-05 |
